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Netherlands Heart Journal
Gepubliceerd in:

01-10-2010 | Interuniversity Cardiology Institute of the Netherlands

Longitudinal arrhythmogenic remodelling in a mouse model of longstanding pressure overload

Auteurs: M. Boulaksil, M. Noorman, M. A. Engelen, T. A. B. van Veen, M. A. Vos, J. M. T. de Bakker, H. V. M. van Rijen

Gepubliceerd in: Netherlands Heart Journal | Uitgave 10/2010

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Extract

Heart failure (HF) is a major burden of disease in industrialised countries with an associated lifetime risk of about 20% in both men and women.1 By definition, heart failure is a functional disease in which cardiac output is unable to meet the demands of the organs. In spite of this, a significant part of mortality in HF patients is caused by sudden cardiac death (SCD), predominantly the outcome of ventricular arrhythmias.1 Nevertheless, mechanisms underlying the evolution of increased vulnerability to arrhythmias during the development of heart failure are not fully comprehended. …
vorige artikel Giant, high pressure pulmonary artery aneurysm in a grand multiparous woman
Literatuur
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Coumel P. The management of clinical arrhythmias. An overview on invasive versus non-invasive electrophysiology. Eur Heart J. 1987;8:92-9.
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Kleber AG, Rudy Y. Basic mechanisms of cardiac impulse propagation and associated arrhythmias. Physiol Rev. 2004;84:431-88.
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Anastasiou-Nana MI, Nanas JN, Karagounis LA, Tsagalou EP, Alexopoulos GE, Toumanidis S, et al. Relation of dispersion of qrs and qt in patients with advanced congestive heart failure to cardiac and sudden death mortality. Am J Cardiol. 2000;85:1212-7.
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Iuliano S, Fisher SG, Karasik PE, Fletcher RD, Singh SN. Qrs duration and mortality in patients with congestive heart failure. Am Heart J. 2002;143:1085-91.
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Marionneau C, Brunet S, Flagg TP, Pilgram TK, Demolombe S, Nerbonne JM. Distinct cellular and molecular mechanisms underlie functional remodeling of repolarizing k+ currents with left ventricular hypertrophy. Circ Res. 2008;102:1406-15.
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Liao Y, Ishikura F, Beppu S, Asakura M, Takashima S, Asanuma H, et al. Echocardiographic assessment of lv hypertrophy and function in aortic-banded mice: Necropsy validation. Am J Physiol Heart Circ Physiol. 2002;282:H1703-1708.
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Barrick CJ, Rojas M, Schoonhoven R, Smyth SS, Threadgill DW. Cardiac response to pressure overload in 129s1/svimj and c57bl/6j mice: Temporal- and background-dependent development of concentric left ventricular hypertrophy. Am J Physiol Heart Circ Physiol. 2007;292:H2119-2130.
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Qu J, Volpicelli FM, Garcia LI, Sandeep N, Zhang J, Marquez-Rosado L, et al. Gap junction remodeling and spironolactone-dependent reverse remodeling in the hypertrophied Heart. Circ Res. 2009;104:365-71.
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Wiegerinck RF, Verkerk AO, Belterman CN, van Veen TA, Baartscheer A, Opthof T, et al. Larger cell size in rabbits with heart failure increases myocardial conduction velocity and qrs duration. Circulation. 2006;113:806-13.
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Allessie MA, Bonke FI, Schopman FJ. Circus movement in rabbit atrial muscle as a mechanism of tachycardia. Iii. The "Leading circle" Concept: A new model of circus movement in cardiac tissue without the involvement of an anatomical obstacle. Circ Res. 1977;41:9-18.
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van Veen TA, Stein M, Royer A, Le Quang K, Charpentier F, Colledge WH, et al. Impaired impulse propagation in scn5a-knockout mice. Combined contribution of excitability, connexin expression, and tissue architecture in relation to aging. Circulation. 2005;112:1927-35.
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Kitamura H, Ohnishi Y, Yoshida A, Okajima K, Azumi H, Ishida A, et al. Heterogeneous loss of connexin43 protein in nonischemic dilated cardiomyopathy with ventricular tachycardia. J Cardiovasc Electrophysiol. 2002;13:865-70.
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Cabo C, Yao JA, Boyden PA, Chen S, Hussain W, Duffy HS, et al. Heterogeneous gap junction remodeling in reentrant circuits in the epicardial border zone of the healing canine infarct. Cardiovasc Res. 2006;72:241-9.
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Peters NS, Coromilas J, Severs NJ, Wit AL. Disturbed connexin43 gap junction distribution correlates with the location of reentrant circuits in the epicardial border zone of healing canine infarcts that cause ventricular tachycardia. Circulation. 1997;95:988-96.
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Boulaksil M, Winckels SK, Engelen MA, Stein M, van Veen TA, Jansen JA, et al. Heterogeneous connexin43 distribution in heart failure is associated with dispersed conduction and enhanced susceptibility to ventricular arrhythmias. Eur J Heart Fail. 2010;12:913-21.
Metagegevens
Titel
Longitudinal arrhythmogenic remodelling in a mouse model of longstanding pressure overload
Auteurs
M. Boulaksil
M. Noorman
M. A. Engelen
T. A. B. van Veen
M. A. Vos
J. M. T. de Bakker
H. V. M. van Rijen
Publicatiedatum
01-10-2010
Uitgeverij
Bohn Stafleu van Loghum
Gepubliceerd in
Netherlands Heart Journal / Uitgave 10/2010
Print ISSN: 1568-5888
Elektronisch ISSN: 1876-6250
DOI
https://doi.org/10.1007/BF03091824

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